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10.1038/srep18132 27
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Growing evidence suggests that an attenuated gut barrier and increased translocation of luminal components into systemic circulation potentially contributes to the aetiology and/or progression of gut localised and systemic ADs (Mu et al
IGF-1 LR3 is better able to stimulate the type 1 IGF receptor and thus induce a higher level of intracellular signaling, cell proliferation and inhibition of apoptosis
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