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A novel biguanide derivative, IM176, induces prostate cancer cell death by modulating the AMPK-mTOR and androgen receptor signaling pathways

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Therefore, current antiinflammatory therapies in PCOS, though not explicitly designed for NET inhibition, may indirectly suppress neutrophil activation, NET formation, and subsequent tissue injuryforming a practical backdoor into the neutrophilNET therapeutic space

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