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This premature polyploidy in Ercc1 -deficient liver is most likely caused by increased levels of p21 in response to accumulating DNA damage (Chipchase, ONeill, and Melton 2003)
Hao Y, Gao X
Alonso A, Gunther K, Maheux-Lacroix S, Abbott J
Furthermore, the upregulation of transforming growth factor (TGF)- and WNT signaling pathways promotes the activation of SNAIL and DLX2, which in turn upregulate the expression of GLS [112]
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