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Although the impact of GLP-1RAs on pancreatic cancer risk remains controversial, emerging evidence suggests that chronic low-grade inflammation and proliferative changes may constitute key pathophysiological mechanisms underlying pancreatic cancer development in patients receiving GLP-1RA therapy [8]

Characterization of this blend is limited to physicochemical profiling, structural verification, and stability-focused investigation conducted exclusively under controlled, non-clinical laboratory conditions

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Mean SEM

Possible interactions between peptides should be expected and tracked