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The resolution of inflammation has emerged as a critical endogenous process that protects host tissues from prolonged or excessive inflammation, which can become chronic

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These findings strongly suggest that the disease-promoting effect of eosinophils does not solely result from their ability to release IL-4 and to elicit a Th2 response, but involves additional mechanisms that support pathogen survival and chronic inflammation

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They found that long-term beta blocker administration decreased endogenous melatonin production by 50%, and in these patients taking the beta blockers, there was a significant increase in sleep latency (the time it takes for a person to fall asleep after lying down in bed and attempting to sleep) and decreased REM sleep (REM sleep is characterized by rapid movement of the eyes, vivid dreaming, and heightened brain activity)