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[DOI] [PMC free article] [PubMed] [Google Scholar] 126.Sourris K.C., Harcourt B.E., Tang P.H., Morley A.L., Huynh K., Penfold S.A., Coughlan M.T., Cooper M.E., Nguyen T.V., Ritchie R.H., et al

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Altogether, these data suggest that the upregulation of GSH, rather than the dTTP pool, is responsible for avoiding oncogene-induced replication stress and less susceptibility to JMF4073 in T315I-Bcr-Abl-32D cells

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