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Thus, using these molecules as templates, we set out to increase metabolic stability and hydrophobicity by incorporating various structural changes that were primarily targeted at the N terminus, the primary site of peptidase-dependent degradation (Abhold and Harding, 1988)

This is because any abnormalities in blood sugar levels exert pressure on the kidneys, causing increased urination, blood in the urine, or even decreased urine production

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For each concentration, the data from 3 trophozoites was analyzed