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NR4a1 overexpression counteracts the ameliorative effect of Sema on pathological myocardial remodeling To provide more convincing evidence that NR4a1 in cardiomyocytes is the target for the effect of Sema on TAC-induced pathological cardiac remodeling, we established a mouse model injected with AAV9-NR4a1

Khaleeluddin K, Philpott WH: I

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The occurrence of hypersensitivity reactions may be increased in patients with renal impairment who receive allopurinol and thiazide diuretics in combination

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