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Certain enteric pathogens possess mechanisms that enable their direct adherence to IECS while others permit the secretion of effector proteins or enterotoxins via membrane localised secretion systems that typically target TJs thereby increasing intestinal permeability by disrupting the intracellular cytoskeleton of IECs (Knig et al

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it does not actively kill all microbes (that would be bactericidal), but it stops them from reproducing, which is sufficient to maintain sterility in a closed multi-dose vial