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Direct stimulation of GLP-1R on cardiomyocytes increases cAMP and activates the PI3K/Akt pathway, thereby improving contractility and reducing apoptosis, which preserves left ventricular function and cardiac output ( , IL-6) and oxidative stress, further protecting the myocardium from inflammatory damage

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Clinical trials have demonstrated reductions in HbA1c (a measure of average blood glucose over three months) with semaglutide treatment, with improvements varying based on baseline levels and dose

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