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BPC-157 and TB-500 also reduce inflammation in animal studies, but as a secondary effect of their tissue-repair pathways, not as a primary mechanism

[1] [3] Primarily, it stimulates insulin secretion from the pancreas when blood glucose levels are elevated, whilst simultaneously suppressing the release of glucagon, a hormone that raises blood sugar

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Is Insulin the Preferred Compound in Lowering Glucose Levels in Patients After a Myocardial Infarction?Reply

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