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Its therapeutic effects are primarily mediated through enhancement of -aminobutyric acid (GABA) activity, inhibition of GABA degradation, and blockade of voltage-gated sodium, calcium and potassium channels [1]

After a complication with my prescribing doctor and insurance, I have been off the medication since May 2025

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Ursodeoxycholic acid and bile-acid mimetics as therapeutic agents for cholestatic liver diseases: An overview of their mechanisms of action