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These animals develop -synuclein aggregation and neuronal dysfunction such as disrupted autophagy [216, 253]

The functions of mammalian cellular clock proteins are additionally regulated through posttranslational modifications through various kinases, methylation, polyadenylation, histone modifications, and non-coding RNAs (Cardone et al., 2005

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The long-term effects of repeated use are not well-characterized in humans