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In endothelial cell models, BPC-157 significantly enhanced ERK1/2 phosphorylation in a dose-dependent manner, leading to increased cellular proliferation, migration, and vascular tube formation mediated through downstream activation of transcription factors c-Fos, c-Jun, and EGR-1

However, most events were mild to moderate and clustered during the first weeks of dose escalation

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Variation in the gut microbiota of laboratory mice is related to both genetic and environmental factors (in Eng)

Owing to its natural resistance to antibiotics, an infection caused by this pathogen can result in serious clinical complications [73]