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Absence of Gclc impairs mitochondrial respiration in FoB Our data above showed that glucose-derived fluxes into the TCA cycle were reduced in GSH-deficient FoB

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Inhibition of mast cell-associated inflammatory pathways in brain injury, stress, and PTSD could be explored as a new therapeutic target to inhibit or prevent the pathogenesis and potentially delay the onset of AD

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The integration of advanced imaging techniques with biomarker analysis promises to reveal new insights into ferroptosis dynamics in vivo, facilitating timely interventions to prevent or reduce brain injury