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Clinical safety studies involving approximately 900 participants showed no clinically significant hormonal changes with AOD-9604, consistent with its peripheral, fat-tissue-specific mechanism of action

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Oxidative stress can result either from the excessive production of reactive oxygen species, which might be anticipated at times of glutamatergic over-activity, or from a reduction in oxidative defences, and can damage cell structures

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What happens after the vote The 8-6 vote is a nonbinding recommendation