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Le Foll, C

Selectively reducing Gsto2 in iSPNs in vivo effectively prevented dopaminergic dysfunction and halted the onset and progression of hyperkinetic symptoms

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LRRK2 plays multiple roles in various signaling pathways via phosphorylation of its substrates [128, 129]

Weve seen what that looks like with compounded GLP-1s and the harm that has come with it the hospitalizations, contaminated products, and in some cases, death, PCAC cannot allow peptides to follow that same path. I don't understand how MAHA can scream about "gold standard" clinical trials for vaccines, but push for the approval of synthetic peptides without proper clinical trials