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However, cancer stem cells are thought to hijack the ability of normal stem cells to express MET, and thus become the cause of cancer persistence and spread to other sites in the body

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In this work, the post-translational modification toward KEAP1 was observed, which subsequently promoted the expression and nuclear translocation of NRF2

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The regulatory effects of miR-138-5p on selenium deficiency-induced chondrocyte apoptosis are mediated by targeting SelM