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The essential link between the mitochondrial carnitine transport and FAO is demonstrated by the life-threatening inherited carnitine/acylcarnitine translocase deficiency (OMIM 613698) that, in contrast to the PCD caused by OCTN2 defects, cannot be compensated by the administration of carnitine (Indiveri et al., 2011)

Because increased oxidative stress is believed to play a central role in the pathogenesis of PD, the association between UA and PD is the most researched field

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