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This redox-dependent interaction mediates immunosuppressive effects through two mechanisms: the suppression of pro-inflammatory signaling pathways and the induction of anti-inflammatory cytokine production [441]

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Liraglutide improved learning and memory performance, attenuated hyperphosphorylation of tau as well as NFs, and prevented neurodegeneration via improving JNK and ERK signaling in brain of 3Tg mice (232)